Population aging represents one of the greatest biomedical and societal challenges of our century. While life expectancy continues to increase worldwide, chronic diseases such as cardiovascular disease, neurodegeneration, pulmonary fibrosis, metabolic disorders and cancer have become major causes of disability and mortality.

Among the mechanisms linking aging to disease, cellular senescence has emerged as a central biological process. Once described as a tumour suppressor programme, senescence is now recognised as a dynamic, heterogeneous cellular state that shapes tissue repair, immune function, inflammation, regeneration and organismal aging.

This Summer School will explore aging from multiple perspectives – from molecular and cellular mechanisms to organ dysfunction, evolutionary theory and innovative therapeutic strategies.

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